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 Guillain - Barre syndrome
Saudi Arabia

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From: "fahad " <hotmail.com> 

To: arabneurosurgery@yahoo.com 

I send this report for a patient having a neurological disease hoping to give me a complete report about availability of his treatment. Its duration program and costs. J hope also to tell me about anyother specialised hospital in the usa that can treat case if the treatment is not available in your hospital . 

Many thanks 
Fahd 
Kingdom of saudi arabia 
Fax 00966

 


KING FAISAL HOSPITAL AND RESEARCH CENTER
NAME OF PATIENT : MAJED 
MEDICAL RECORD NUMBER: 361977 

      DIAGOSIS:  Guillain - Barre syndrome

      The patient is a 21- year- old Saudi male who was admitted in April 1995 to King Faisal Specialist Hospital & research center and diagnosed with acute Guillain – Barre syndrome. At that time he had a history of weakness that started while playing football. A few hours later he developed calf pain and then had an ascending paraalysis that led to quadriplegia. The patient was admitted to the hospital and underwent evaluation and was diagnosed with acute Guillain – Barre syndrome and treated. The past medical history at that time was significant for a probable diagnosis of polio at age five. Since that time, the patient improved and became able to walk independently. His nerve conduction improved.
      I began seeing this patient on 14 May 1997 and at that time the patient reported that he was ambulating independently, however he had residual distal weakness. His motor examination at that time revealed a deltoid strength of 5, biceps 5, triceps 5, wrist extensors 4, wrist flexors 4, finger flexors 4, iliopsoas 5, quadriceps 5, tibialis anterior 1, gastrocnemius 4, extensor hallucis longus 1. The patient at that time was diagnosed with vitamin D deficiency and vitamin D replacment therapy was instituted.
      The patient was most recently seen today and is more or less stable. He feels now that with the cold weather, his handwriting is worse and he has some pain in his left leg. His examination is otherwise unchanged.
      The patient’s father and I discussed outside referrals for a cure for this disorder, and I explained to him the diagnosis of Guillain – Barre and suggested that we repeat nerve conduction studies. The father agreed with this approach. My plan is to schedule him for an outpatient electromyography and nerve conduction studies, and to follow him up in my clinic afterwards to discuss the results of this study with the patient and his father.
      Note: This medical report is being dictated for patient care purposes and does not constitute a referral.
       
       

                             SIGNATURE -----------------------------------------------------------------
                                                     Edward James Cupler, MD
                                                      Consultant Neurologist
                                                      Diplomate, American Board of Neurology ( U.S.A)
      Department of Neurosciences
      D: 13 December 1998
      T: 26 December 1998/fg
      C:       Department of Neuroscienes
                  Dr . Mohammed Ashfaq Ezad, King Saud hospital, Unaizah (MRN: 78371)
                  Dr . E . Cupler

       



       

      KING DOM OF SAUDI ARABIA
      MINISTRY OF Health
      General Directorate of Health Affairs, Gassim
      KFSH
       Therapeutic Services Department
       

      Date: 11/04/1422  ( 02/07/2001 )  MEDICAL REPORT
      NAME OF THE PATIENT: MAGID 
      AGE/ SEX: 22 YEARS OLD / MALE 
      SAUDI  NATIONALTY
      DIAGNOSIS: - GUILLAN BARRE SYNDROME

      This 22-year-old Saudi male patient was seen in the outpatients clinic with the above-mentioned diagnosis for review.

      His condition started in the year 1415 when the had subacute severe weakness of both upper and lower limbs following 3 days of respiratory illness.
      He was initially complaining of paraesthenal climbing stairs. He was followed up over the last 5 years in King Faisal specialist hospital & RC, Riyadh. The initial severe weakness improved but condition remained static with no further improvement of his leg weakness.

      On Examination:
      Patient fully conscious, thin built with wasting of all upper and lower limb muscles particularly distal ones.
      Power was only mildly impaired in proximal upper limb muscles but lower limb proximal and distal muscles are weak with zero grade of power in ankle dorsiflexors and plantar flexors.
      Sensations intact. Reflexes depressed. Plantar bilaterally flexor.

      Conclusion / Recommendations:
      The patient is still disabled by effects of his neuropathy and is in need for further follow up and physiotherapy.

      DR. EL GAMRI E. MOHD 
      DR. HESHAM NADRAH
      Consultant Neurologist/ Chief of Medicine 
      Hospital Director
       


 
 
 
 
 

 

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